Atkins and Athletes

Former Member
Former Member
Some time ago there was a thread about Atkins diet and exercise. I am wondering if anyone has some good advice about using this diet when you are an athlete. I work out every day, whether it's 4,000 yards in the pool, or 3-4 miles on the treadmill, or dryland training (weights). I went on Atkins a week ago to lose 10 pounds, so far I've lost 4 and feel terrific. Last Saturday I had a 5000-yard workout that was the best in ages. So much energy! I'm just wondering if it's because I've added much-needed protein to my diet (I'm a carb junkie). A fellow athlete poo-poo's the Atkins plan and says it's a bunch of hogwash, that athletes HAVE to have some carbs and they won't kill you. Anyone?
Parents
  • Former Member
    Former Member
    Cynthia, You are pretty good on your assumptions. Those are part of the reasons. We can tract cow's milk consumption in all regions of europe and the world. There are large parts of Germany that did not drink cows milk. Up till the 1930's the largest immigrant group in the USA was Germans. Many Germans do not have the ability to digest the lactose in milk. Those like myself have a recessive gene ( I am half german - half scottish -all american) GALT enzyme (the enzyme needed to convert galactose into a form useable by the body The body breaks the lactose down into galactose and glucose. Glucose is the sugar used by the body for energy. Galactosemia means too much galactose in the blood caused by the individual "missing" the enzyme (known as GALT) to convert galactose into glucose. This accumulation of galactose is a poison to the body and can cause serious complications such as the following and if untreated, as high as 75% of infants will die: Remember seeing the photos of "starving" african children ater WW2 when the good old USA was sending ships full of powdered milk? The childrens bellies were bloated, but not from starvation. Those were the effects of milk on people who could not digest it.:mad: "Milk is NOT good for everybody". I ended up marrying a beauty queen winner from the Phillipines, both of us are recessive carriers of classic galactosemia, inheriting one normal gene from one parent and one gene containing the error that leads to classic galactosemia from the other parent. This person's genotype would be G/N and their enzyme activity would be less than normal, but not so much so as to cause medical complications or require dietary management. Out of four kids, one has galactosemia. Hence my knowledge of milk consumption. At the end of 1998, cases of galactosemia were recorded in 24 different populations and ethnic groups in 15 countries worldwide. The mutations most frequently cited are Q188R, K285N, S135L, and N314D. Q188R is the most common mutation in European populations or in those predominantly of European descent. A small ethnic group in Ireland, known as The Travelers, can be as much as 60 times as prone to the Q188R mutation as is the general Irish population. Genetics has many things I enjoy, history, geography, chemistry, all together. If you or anyone else is interested, look at http://www.galactosemia.org
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  • Former Member
    Former Member
    Cynthia, You are pretty good on your assumptions. Those are part of the reasons. We can tract cow's milk consumption in all regions of europe and the world. There are large parts of Germany that did not drink cows milk. Up till the 1930's the largest immigrant group in the USA was Germans. Many Germans do not have the ability to digest the lactose in milk. Those like myself have a recessive gene ( I am half german - half scottish -all american) GALT enzyme (the enzyme needed to convert galactose into a form useable by the body The body breaks the lactose down into galactose and glucose. Glucose is the sugar used by the body for energy. Galactosemia means too much galactose in the blood caused by the individual "missing" the enzyme (known as GALT) to convert galactose into glucose. This accumulation of galactose is a poison to the body and can cause serious complications such as the following and if untreated, as high as 75% of infants will die: Remember seeing the photos of "starving" african children ater WW2 when the good old USA was sending ships full of powdered milk? The childrens bellies were bloated, but not from starvation. Those were the effects of milk on people who could not digest it.:mad: "Milk is NOT good for everybody". I ended up marrying a beauty queen winner from the Phillipines, both of us are recessive carriers of classic galactosemia, inheriting one normal gene from one parent and one gene containing the error that leads to classic galactosemia from the other parent. This person's genotype would be G/N and their enzyme activity would be less than normal, but not so much so as to cause medical complications or require dietary management. Out of four kids, one has galactosemia. Hence my knowledge of milk consumption. At the end of 1998, cases of galactosemia were recorded in 24 different populations and ethnic groups in 15 countries worldwide. The mutations most frequently cited are Q188R, K285N, S135L, and N314D. Q188R is the most common mutation in European populations or in those predominantly of European descent. A small ethnic group in Ireland, known as The Travelers, can be as much as 60 times as prone to the Q188R mutation as is the general Irish population. Genetics has many things I enjoy, history, geography, chemistry, all together. If you or anyone else is interested, look at http://www.galactosemia.org
Children
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